|Description||ACTIVE HALF-LIFE 5 hours
TABLET COUNT: 100 COUNTS
CLASSIFICATION Anabolic Steroid
DOSAGE Men 5-20 mg/day
WATER RETENTION No
AROMATIZATION No||Active Ingredient: Mesterolone 10mg
Bottle Count: 50 Tablets
Active Half-life: 12 Hours
Classification: Anabolic Steroid
Dosage Men: 30-100 mg/day
Water Retention: No
Concentration: 10 mg/Pill
Presentation: 50 Tablets (Total box 500 mg)
Dosage: 30-100mg per day over the course of 8-12 weeks||Active Ingredient: Methandienone 10mg
Active Half-life: 5-6 Hours
Classification: Anabolic Steroid
Concentration: 10 mg/tab
Presentation: 100 Tablets (Total box 1000mg)
Dosage: 20-80mg per day over the course of 6-8 weeks||Tablet Count: 100 tablets
Dose: 20– 60mcg per day
Cycle: 6-8 weeks.
Bioavailability: 89–98% (orally)
Molar mass: 277.19
Elimination half-life: 36–48 hours
Metabolism: Hepatic (negligible)
Water Retention: RARELY
Aromatization: YES||Warehouse : EU 3(Dues Medical)
Active Ingredient: Oxymetholone 50mg
Tablet Count: 50 Tabs
Concentration: 50 mg/Tab
Presentation: 50 Tablet (Total box 2500mg)
Dosage: 50-150mg per day over the course of 4-6 weeks
Trade name: Anadrol, Anadromed, others
ATC code: A14AA05 (WHO)||ACTIVE INGREDIENT: Exemestane 25 mg
TABLET COUNT: 30 COUNTS
ACTIVE HALF-LIFE: 9 HOURS
CLASSIFICATION: ANABOLIC STEROID
DOSAGE MEN: 25-50 MG/DAY
DOSAGE WOMEN: 5-30 MG/DAY
WATER RETENTION: NO
Ship From: Europe|
|Content||M-1-T (or Methyldihydroboldenone) – powerful testosterone derivative, active on oral administration. Methyldihydroboldenone was obtained in the 1950s, at the beginning of the study of steroids. However, this steroid, as well as most of the others, did not go into serial production. This steroid has signs of other 1-enes of Primobolan structure, the ability of oral absorption due to 17a methyl group like Winstrol and high ability to reach the receptors, like Trenbolone.
But Methyl-1-Testosterone capabilities exceed the capabilities of any medical steroid sold in our time. As a Primobolan derivative – Methyldihydroboldenone is not flavored. Normally this would mean that it builds high-quality hard muscles, but not the majority of those who used M-1-T reported significant water retention, even when used in moderation. This shows a moderate progestogenic activity of the drug. Progestogenic steroids can increase the effects of estrogen, so is not desirable to use it in conjunction with flavoring steroids, and it is recommended to use anti-estrogens such as Nolvadex. You can also expect the standard androgenic side effects (greasiness, acne, hair loss).
Because of its structure, M-1-T it is an equilibrated steroid, anabolic effect which is greater than androgen.
Since it is 17 alpha-methylated, you need to give serious consideration to its side effects, liver toxicity. Due to the fact that it is so powerful, the problems will be bigger than that of other oral drugs.
Perhaps you think that 20 mg of M-1-T toxicity will be equal to 20mg of Dianabol toxicity? No, M-1-T stronger harms the liver, so it is advisable to use it at a dose of 5-10mg, for very advanced bodybuilders – 20 mg per day for 6-8 weeks. Most best will be to use M-1-T with a dose of 10 mg in conjunction with Testosterone Cypionate 400 mg per week. This will allow to balance the anabolic and androgenic effects and minimize the harm on the liver.
Warnings: Keep out of reach of children. For adults only.||Methandienone 10mg x 100 tablets
Methandienone was used as a stimulating recovery processes, revitalizing, treating burns tools. Today, the use of Methandrostenolone athletes to increase muscle mass.Steroid profile increase in muscle mass due to stimulating the production of protein. A significant part of gaining weight is a persons in the body of water. If you do not take appropriate pharmacological agents, most often occurs the phenomenon of recoil.||Clenbuterol hydrochloride 0.02mg x 100 tablets
Clenbuterol is a widely used bronchodilator in many parts of the world. The drug is most often prepared in 20mcg tablets, but it is also available in syrup and injectable form. Clenbuterol belongs to a broad group of drugs knows as sympathomimetics.||Exemestane is a synthetic androgen analogue. It’s is a steroidal inhibitor of aromatase which binds irreversibly to and inhibits the enzyme aromatase, thereby blocking the conversion of cholesterol to pregnenolone and the peripheral aromatization of androgenic precursors into estrogens.
Exemestane has been associated with a low rate of serum enzyme elevations during therapy and rare instances of clinically apparent liver injury.
This medication is used to treat certain types of breast cancer (such as hormone-receptor-positive breast cancer) in women after menopause. Exemestane is also used to help prevent the cancer from returning. Some breast cancers are made to grow faster by a natural hormone called estrogen. Exemestane decreases the amount of estrogen the body makes and helps to slow or reverse the growth of these breast cancers. Exemestane is usually not used in women of childbearing age.
Exemestane is usually taken once a day after a meal. Take Exemestane at around the same time every day. You may need to take Exemestane for several years or longer. Continue to take Exemestane even if you feel well. Do not stop taking Exemestane without talking to your doctor.
Effect On Estrogens
Plasma estrogen (estradiol, estrone, and estrone sulfate) suppression is seen starting at a 5-mg daily dose of Exemestane, with a maximum suppression of at least 85% to 95% achieved at a 25-mg dose. Exemestane 25 mg daily reduces whole body aromatization (as measured by injecting radiolabeled androstenedione) by 98% in postmenopausal women with breast cancer. After a single dose of Exemestane 25 mg, the maximal suppression of circulating estrogens occurres 2 to 3 days after dosing and persisted for 4 to 5 days.
Effect On Corticosteroids
In multiple-dose trials of doses up to 200 mg daily, Exemestane selectivity was assessed by examining its effect on adrenal steroids. Exemestane did not affect cortisol or aldosterone secretion at baseline or in response to ACTH at any dose. Thus, no glucocorticoid or mineralocorticoid replacement therapy is necessary with Exemestane treatment.
Other Endocrine Effects
Exemestane does not bind significantly to steroidal receptors, except for a slight affinity for the androgen receptor (0.28% relative to dihydrotestosterone). The binding affinity of its 17dihydrometabolite for the androgen receptor, however, is 100 times that of the parent compound.
Daily doses of Exemestane up to 25 mg had no significant effect on circulating levels of androstenedione, dehydroepiandrosterone sulfate, or 17-hydroxyprogesterone, and were associated with small decreases in circulating levels of testosterone.
Increases in testosterone and androstenedione levels have been observed at daily doses of 200 mg or more. A dose-dependent decrease in sex hormone binding globulin (SHBG) has been observed with daily Exemestane doses of 2.5 mg or higher.
Slight, nondose-dependent increases in serum luteinizing hormone (LH) and follicle-stimulating hormone (FSH) levels have been observed even at low doses as a consequence of feedback at the pituitary level. Exemestane 25 mg daily had no significant effect on thyroid function [free triiodothyronine (FT3), free thyroxine (FT4), and thyroid stimulating hormone (TSH).
Exemestane is distributed extensively into tissues. Exemestane is 90% bound to plasma proteins and the fraction bound is independent of the total concentration. Albumin and α11-acid glycoprotein both contribute to the binding. The distribution of Exemestane and its metabolites into blood cells is negligible.
Exemestane is extensively metabolized. The initial steps in the metabolism of Exemestane are oxidation of the methylene group in position 6 and reduction of the 17-keto group with subsequent formation of many secondary metabolites.
Each metabolite accounts only for a limited amount of drug-related material. The metabolites are inactive or inhibit aromatase with decreased potency compared with the parent drug. One metabolite may have androgenic activity [see Pharmacodynamics]. Studies using human liver preparations indicate that cytochrome P 450 3A4 (CYP 3A4) is the principal isoenzyme involved in the oxidation of Exemestane. Exemestane is metabolized also by aldoketoreductases.|
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