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Turanaxyl – Kalpa Pharmaceuticals (INT)
$50.00
Availability: In StockCHLORODEHYDROMETHYLTESTOSTERONE 10 MG TABS
Drug Class: Androgen; Anabolic Steroid
Composition:
– Active Substance: Chlorodehydromethyltestosterone
– Concentration: 10 mg/tab
Presentation: 100 tabs
Manufacturer: Kalpa Pharmaceuticals
Common Name(s): Oral Turinabol, Tbol, Turanabol
Quick Comparison
| Settings | Turanaxyl - Kalpa Pharmaceuticals (INT) remove | Letrozole 2.5mg – Anti-estrogen, decreases fat build-up and decreases water retention remove | Clomid 50mg – Clomiphene Citrate for Anti Estrogen, block Estrogen remove | Anadromed 50mg – Oxymetholone – Deus Medical remove | Turimed 10mg – Turinabol – Deus Medical remove | Exemestane 25mg - Hilma Biocare remove |
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| Name | Turanaxyl - Kalpa Pharmaceuticals (INT) remove | Letrozole 2.5mg – Anti-estrogen, decreases fat build-up and decreases water retention remove | Clomid 50mg – Clomiphene Citrate for Anti Estrogen, block Estrogen remove | Anadromed 50mg – Oxymetholone – Deus Medical remove | Turimed 10mg – Turinabol – Deus Medical remove | Exemestane 25mg - Hilma Biocare remove |
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| SKU | kalpa-int-006 | 101AAS-HB-0-139 | 101AAS-HB-0-129 | 101AAS-DM-0-112 | 101AAS-DM-0-103 | 101AAS-HB-0-138 |
| Rating | ||||||
| Price | $50.00 | $75.00 | $75.00 | $65.98 | $60.00 | $90.00 |
| Stock | Out of stock | Out of stock | Out of stock | |||
| Availability | Out of stock | Out of stock | Out of stock | |||
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| Description | CHLORODEHYDROMETHYLTESTOSTERONE 10 MG TABS Drug Class: Androgen; Anabolic Steroid Composition: - Active Substance: Chlorodehydromethyltestosterone - Concentration: 10 mg/tab Presentation: 100 tabs Manufacturer: Kalpa Pharmaceuticals Common Name(s): Oral Turinabol, Tbol, Turanabol | CTIVE INGREDIENT: Letrozole 2.5mg TABLET COUNT: 30 COUNTS ACTIVE HALF-LIFE: 9 HOURS CLASSIFICATION: ANABOLIC STEROID DOSAGE: 2.5-5 MG/DAY ACNE: RARELY WATER RETENTION: NO HBR: NO HEPATOXITY: LOW AROMATIZATION: NO Ship From: Europe | Active Ingredient: Clomiphene Citrate (Clomid) Tablet Count: 50 Tab Molar Mass: 405.966 G/mol Formula: C26h28clno Trade Name: Clomid Concentration: 50 mg/cap Presentation: 50 Tablets (Total box 2500mg) Ship From: Europe | Warehouse : EU 3(Dues Medical) Active Ingredient: Oxymetholone 50mg Tablet Count: 50 Tabs Concentration: 50 mg/Tab Presentation: 50 Tablet (Total box 2500mg) Dosage: 50-150mg per day over the course of 4-6 weeks Trade name: Anadrol, Anadromed, others Metabolism: Liver ATC code: A14AA05 (WHO) | Active Ingredient: Chlorodelydromethytestosterone 10mg Tablet Count: 50 Counts Active Half-life: 9 Hours Classification: Anabolic Steroids Dosage Men: 10mg /day Dosage Women: 10mg/day Acne: Rarely Water Retention: No Hbr: No Hepatoxity: Low Aromatization: Yes | ACTIVE INGREDIENT: Exemestane 25 mg TABLET COUNT: 30 COUNTS ACTIVE HALF-LIFE: 9 HOURS CLASSIFICATION: ANABOLIC STEROID DOSAGE MEN: 25-50 MG/DAY DOSAGE WOMEN: 5-30 MG/DAY ACNE: RARELY WATER RETENTION: NO HBR: NO HEPATOXITY: LOW Ship From: Europe |
| Content | Letrozole is a nonsteroidal inhibitor of estrogen synthesis with antineoplastic activity. As a third-generation aromatase inhibitor, Letrozole selectively and reversibly inhibits aromatase, which may result in growth inhibition of estrogen-dependent breast cancer cells. Aromatase, a cytochrome P-450 enzyme localized to the endoplasmic reticulum of the cell and found in many tissues including those of the premenopausal ovary, liver, and breast, catalyzes the aromatization of androstenedione and testosterone into estrone and estradiol, the final step in estrogen biosynthesis. Estrogens are produced by the conversion of androgens through the activity of the aromatase enzyme. Letrozole blocks production of estrogens in this way by competitive, reversible binding to the heme of its cytochrome P450 unit. The action is specific, and letrozole does not reduce production of mineralo- or corticosteroids. In contrast, the antiestrogenic action of tamoxifen, the major medical therapy prior to the arrival of aromatase inhibitors, is due to its interfering with the estrogen receptor, rather than inhibiting estrogen production. Letrozole is approved by the United States Food and Drug Administration (FDA) for the treatment of local or metastatic breast cancer that is hormone receptor positive or has an unknown receptor status in postmenopausal women. Side effects include signs and symptoms of hypoestrogenism. There is concern that long term use may lead to osteoporosis, which is why prescriptions of Letrozole are often accompanied by prescriptions of osteoporosis-fighting medication such as Fosamax. Letrozole has shown to reduce estrogen levels by 98 percent while raising testosterone levels. The anti-estrogen action of letrozole is preferred by athletes and bodybuilders for use during a steroid cycle to reduce bloating due to excess water retention and prevent the formation of gynecomastia related breast tissue that is a side effect of some anabolic steroids. Usage above 2.5 mg/day is known to potentially temporarily kill sex drive. Above 5mg/day for extended periods may cause kidney problems. Letrozole has also been shown to delay the fusing of the growth plates in adolescents. This may boost the effectiveness of growth hormone, and thus Letrozole is used to treat adolescents and children with short stature. | Clomiphene – Post Cycle Therapy
The basic need in bodybuilding is the right amount of testosterone secretion of an athlete. It enables them to perform and also gaining the ripped structure. Preference for the testosterone is on top. The testosterone level can be maintained by a right regimen and also with the help of some steroids. One of them is Clomiphene, which can also be quoted as recoveree for testosterone. If the level of testosterone is not up to the mark or the bodybuilder need more push, then it should be used.
What is Clomiphene?
Clomid is extensively used for the enhancement of testosterone production. It takes a vital place after anabolic steroid cycles. It is the next step after taking anabolic steroids.
It works by taking the space of binding sites of estrogen receptors without activating. If the estrogen levels will be low then there is a supposition for hypothalamus. Hypothalamus is gland beneath the brain which secretes hormones.
Generally, Clomiphene dosage is at 50mg/day. It has a long life. The body contains the Clomipheneof past dosage, simultaneously keeping the present dose of any bodybuilder. It takes time to get going. For example, if you are taking Clomiphene from last week, and today you have taken the dose, your body also has that amount of past Clomiphenein the system. It’s fine if the blood level is right.
Benefits
| Exemestane is a synthetic androgen analogue. It’s is a steroidal inhibitor of aromatase which binds irreversibly to and inhibits the enzyme aromatase, thereby blocking the conversion of cholesterol to pregnenolone and the peripheral aromatization of androgenic precursors into estrogens. Exemestane has been associated with a low rate of serum enzyme elevations during therapy and rare instances of clinically apparent liver injury. This medication is used to treat certain types of breast cancer (such as hormone-receptor-positive breast cancer) in women after menopause. Exemestane is also used to help prevent the cancer from returning. Some breast cancers are made to grow faster by a natural hormone called estrogen. Exemestane decreases the amount of estrogen the body makes and helps to slow or reverse the growth of these breast cancers. Exemestane is usually not used in women of childbearing age. Exemestane is usually taken once a day after a meal. Take Exemestane at around the same time every day. You may need to take Exemestane for several years or longer. Continue to take Exemestane even if you feel well. Do not stop taking Exemestane without talking to your doctor. Effect On Estrogens Plasma estrogen (estradiol, estrone, and estrone sulfate) suppression is seen starting at a 5-mg daily dose of Exemestane, with a maximum suppression of at least 85% to 95% achieved at a 25-mg dose. Exemestane 25 mg daily reduces whole body aromatization (as measured by injecting radiolabeled androstenedione) by 98% in postmenopausal women with breast cancer. After a single dose of Exemestane 25 mg, the maximal suppression of circulating estrogens occurres 2 to 3 days after dosing and persisted for 4 to 5 days. Effect On Corticosteroids In multiple-dose trials of doses up to 200 mg daily, Exemestane selectivity was assessed by examining its effect on adrenal steroids. Exemestane did not affect cortisol or aldosterone secretion at baseline or in response to ACTH at any dose. Thus, no glucocorticoid or mineralocorticoid replacement therapy is necessary with Exemestane treatment. Other Endocrine Effects Exemestane does not bind significantly to steroidal receptors, except for a slight affinity for the androgen receptor (0.28% relative to dihydrotestosterone). The binding affinity of its 17dihydrometabolite for the androgen receptor, however, is 100 times that of the parent compound. Daily doses of Exemestane up to 25 mg had no significant effect on circulating levels of androstenedione, dehydroepiandrosterone sulfate, or 17-hydroxyprogesterone, and were associated with small decreases in circulating levels of testosterone. Increases in testosterone and androstenedione levels have been observed at daily doses of 200 mg or more. A dose-dependent decrease in sex hormone binding globulin (SHBG) has been observed with daily Exemestane doses of 2.5 mg or higher. Slight, nondose-dependent increases in serum luteinizing hormone (LH) and follicle-stimulating hormone (FSH) levels have been observed even at low doses as a consequence of feedback at the pituitary level. Exemestane 25 mg daily had no significant effect on thyroid function [free triiodothyronine (FT3), free thyroxine (FT4), and thyroid stimulating hormone (TSH). Absorption Exemestane is distributed extensively into tissues. Exemestane is 90% bound to plasma proteins and the fraction bound is independent of the total concentration. Albumin and α11-acid glycoprotein both contribute to the binding. The distribution of Exemestane and its metabolites into blood cells is negligible. Metabolism Exemestane is extensively metabolized. The initial steps in the metabolism of Exemestane are oxidation of the methylene group in position 6 and reduction of the 17-keto group with subsequent formation of many secondary metabolites. Each metabolite accounts only for a limited amount of drug-related material. The metabolites are inactive or inhibit aromatase with decreased potency compared with the parent drug. One metabolite may have androgenic activity [see Pharmacodynamics]. Studies using human liver preparations indicate that cytochrome P 450 3A4 (CYP 3A4) is the principal isoenzyme involved in the oxidation of Exemestane. Exemestane is metabolized also by aldoketoreductases. | |||
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